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From: jay on 6 Aug 2008 16:02
> There is NO correlation between death, disease or disorders and the
> amounts of pesticides present in the American food supply.

Cancer of the mouth and pharynx, occupation and exposure to chemical
agents in Finland [in 1971-95].

The objective of this article was to find associations between cancer
of the mouth and pharynx, occupation and chemical exposure. A cohort
of Finns born between 1906 and 1945 was followed-up for 46.8 (21.5 in
males and 25.3 in females) million person-years during 1971-95.
Incident cases of cancer of the mouth and pharynx (n = 2,708) were
identified in a record linkage with the Finnish Cancer Registry. The
Census occupations in 1970 were converted to chemical exposures with a
job-exposure matrix (FINJEM). Cumulative exposure (CE) was calculated
as the product of prevalence, level and duration of the exposure.
Standardized incidence ratio (SIR) was calculated for each of the 393
occupations, and for CE categories of the 43 chemical agents, using
total Finnish population as reference. Relative risks (RR) comparing
various CE-categories with unexposed ones were defined for selected
agents by Poisson regression analysis. Elevated SIRs were observed
among lawyers, authors, journalists, performing artists, musicians,
electronics and telefitters, painters (building), building hands,
dockers, unskilled labourers and hotel porters in males and private
secretaries, dressmakers, shoemakers and cobblers, waiters, pursers
and stewardesses in females. The multivariate analyses showed high RRs
for high exposure to aliphatic and alicyclic hydrocarbons, pesticides
and alcohol. In conclusion, occupations with high SIRs were mostly the
ones with high consumption of alcohol. Exposure to solvents and
possibly to pesticides, engine exhaust, textile dust and leather dust
may increase the risk of cancer of mouth and pharynx. PMID: 18470913
From: jay on 6 Aug 2008 16:07
On Aug 6, 1:39 pm, Always Learning <noPublicEm...(a)this.time> wrote:
> On Wed, 6 Aug 2008 11:07:05 -0700 (PDT), jay <jaym1...(a)hotmail.com>
> wrote this stuff here :
>
> >Below is a list of Fruits & Veggies
> >with the most pesticides based on U.S. FDA data collected between 2000
> >and 2005 fromhttp://www.foodnews.org
>
> There is NO correlation between death, disease or disorders and the
> amounts of pesticides present in the American food supply.

Persistent organochlorine pesticides and risk of testicular germ cell
tumors.

BACKGROUND: Exposure to endocrine-disrupting chemicals, such as
persistent organochlorine pesticides, has been suggested to increase
the risk of testicular germ cell tumors (TGCTs) ... CONCLUSIONS:
Increased exposure to p,p'-DDE may be associated with the risk of both
seminomatous and nonseminomatous TGCTs, whereas exposure to chlordane
compounds and metabolites may be associated with the risk of seminoma.
Because evidence suggests that TGCT is initiated in very early life,
it is possible that exposure to these persistent organic pesticides
during fetal life or via breast feeding may increase the risk of TGCT
in young men. PMID: 18445826
From: jay on 6 Aug 2008 16:11
> There is NO correlation between death, disease or disorders and the
> amounts of pesticides present in the American food supply.

Pesticide residues and reproductive dysfunction in different
vertebrates from north India.

Organochlorines (isomers of hexachlorocyclohexane--HCHs and
metabolites of dichlorodiphenyltrichloroethane--DDTs, aldrin and
endosulfan) and organophosphate (chlorpyrifos) insecticide residues
were investigated by gas liquid chromatography in the blood of fish,
chick, goat and man ... Our results indicated that increase of
insecticides in blood level in vertebrates causes reproductive
dysfunction and suggested that for human beings food like fish, chick
and goat containing beyond permissible limit of insecticides must be
avoided. PMID: 18499323
From: jay on 6 Aug 2008 16:29
> There is NO correlation between death, disease or disorders and the
> amounts of pesticides present in the American food supply.

Effects of model organophosphorous pesticides on DNA damage and
proliferation of HepG2 cells.

Organophosphorous compounds (OPs) are commonly used pesticides. The
primary mechanism of OP toxicity is the inhibition of acetylcholine
esterase in the nervous system leading to a variety of acute and
chronic effects. Recent studies have revealed several other targets of
OPs that disturb noncholinergic biological systems. We investigated
whether low concentrations of model OPs-methyl parathion (PT), methyl
paraoxon (PO), and dimefox (DF)-induce DNA damage and/or affect cell
proliferation in human hepatoma HepG2 cells ... PT induced DNA damage
at lower concentrations (1 microg/mL) than PO (100 microg/mL), whereas
DF did not induce DNA damage. PT and PO caused a reduction of cell
proliferation at their highest concentrations (100 microg/mL), while
DF increased cell proliferation at all concentrations used (0.01-100
microg/mL). PT and PO upregulated expression of DNA damage responsive
genes ... We conclude that PT and PO are genotoxic, while DF shows
mitogenic activity. An important finding of this study is that PT had
higher genotoxic potential than PO, which warrants for further
investigations to correctly evaluate the hazards of exposure to these
chemicals. PMID: 18418871
From: jay on 6 Aug 2008 16:31
> There is NO correlation between death, disease or disorders and the
> amounts of pesticides present in the American food supply.

Cell adhesion proteins altered by 17beta estradiol and parathion in
breast epithelial cells.

The association between breast cancer initiation and prolonged
exposure to estrogen suggests that this hormone may also have an
etiologic role in such a process. On the other hand, many studies have
found an association between human cancer and exposure to agricultural
pesticides such as parathion, an organophosphorous pesticide used in
agriculture to control mosquito plagues. However, the key factors
behind the initiation of breast cancer remain to be elucidated. The
aim of this study was to determine the effect of 17beta estradiol
(estrogen) and parathion on protein expression in cell transformation
of human breast epithelial cells in vitro. Estrogen and parathion
alone and in combination induced malignant transformation of an
immortalized human breast epithelial cell line, MCF-I0F, as indicated
by anchorage independency and invasive capabilities. The results
indicate that a combination of estrogen and parathion increased the
expression of related cell adhesion proteins such as Dvl, Notch, CD146
and beta catenin. In conclusion, it can be suggested that pesticides
affect human breast cell adhesion changes indicative of
transformation. PMID: 18097591
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